To further study whether ARHGAP9 expression was influenced by different chromosomal abnormalities, we retrieved two microarray data (GSE14468 and GSE13159) from the GEO database and evaluated ARHGAP9 expression among AML patients with major recurrent chromosomal translocations, including inv(16), t(8;21), t(15;17), 11q23, and complex, as well as the normal karyotype. This evidence concerns the gene ARHGAP9 and acute myeloid leukemia.