To determine whether the increased angiogenic potential of NSCLC cells depleted of TLR7 could be due to their defective pro-resolving pathways’ expression, we assessed the effects of exogenously added SPMs (RvD1 and LXB4) on pro-angiogenic activity of NSCLC cells silenced for TLR7. Here, TLR7 is linked to non-small cell lung carcinoma.