SOAT1 and infection: With regard to immune genes, several genes in the Toll (e.g., GNBP3, Serpin-27A, and Drosomycin) and Imd (e.g., PGRP-LB, Kenny, Relish, and Attacin) pathways and fewer genes in the Janus kinase (Jak)-signal transducer and activator of transcription (Stat) (Jak/Stat) (e.g., Tot-A) and c-Jun N-terminal kinase (Jnk) (e.g., puckered) pathways as well as genes encoding factors that participate in cellular immune activities (e.g., Tep1, Tep2, and hemese) were differentially affected throughout the infection [5,38,39,40].