IL-17-producing CD4+ and IL-17+ γδ+T cells are also detected in the plaques, where they likely support the development of atherosclerosis via the regulation of myeloid cell migration to the aorta and provide plaque stability via TGFβ-dependent mechanisms; however, other studies also suggest a protective role of Th17 cells [13]. The gene discussed is TGFB1; the disease is atherosclerosis.