miR-17-92 downregulated multiple components of TGFβ signaling to evade cell cycle arrest and/or apoptosis in neuroblastoma [72], and upregulation of miR-17-92 by c-Myc caused downregulation of TGFβ signaling pathway components to stimulate angiogenesis and tumor growth [73]. Here, TGFB1 is linked to neuroblastoma.