During sepsis, bacterial surface molecules, such as lipopolysaccharides (LPS), may evoke an inflammatory catastrophe with outbursts of inflammatory cytokines, including interleukin-6 (IL-6), macrophage inflammatory protein-2 (MIP-2), and tumor necrosis factor alpha (TNF-α), as well as vascular endothelial growth factor (VEGF), which can cause epithelial and endothelial damage, vascular leakage, lung edema, and hypoxemia, eventually resulting in multiple organ system failure [1,2,3,4,5,6]. The gene discussed is IL6; the disease is Sepsis.