IGF1 and multiminicore myopathy: Similarly, hsa-miR-6722-3p/-328-3p, interacting with endothelial cell by having an impact on STAT3 (signal transducer and activator of transcription 3,) IGF-1 (insulin Like Growth Factor 1), and PTEN (phosphatase and tensin homolog)-signaling pathways, were abnormally upregulated in an MMD-discordant, monozygotic twin cohort.