SYNPO2 and triple-A syndrome: Focal subsarcolemmal and sarcoplasmic increase of Synaptopodin-2 not only indicates an impact of this protein (which is also involved in the modulation of autophagy; [38] in the molecular myopathology of Allgrove syndrome but also accords with our proteomic findings obtained in both proteomic studies on skin fibroblasts derived from the same patient.