Recently, recurrent mutations affecting KRAS and PIK3CA have been reported in ovarian endometriosis, deep infiltrative endometriosis, uterine adenomyosis, normal endometrium, and iatrogenic endometriosis (i.e., extragonadal endometriosis), suggesting that driver mutations of oncogenes are likely to exist in RSEACs [27,28,29,30,31,32,33,34,35]. The gene discussed is KRAS; the disease is ovarian endometriosis.