Furthermore, ASM cells derived from biopsies of both mildly and moderately asthmatic lungs were shown to have increased secretion of eotaxin compared with control ASM cells, and given that this chemokine is a platelet chemoattractant with relevance to asthma and has been reported elsewhere in sputum and BAL fluid (44–46), this increase provides a link between patient-derived data and the mechanistic understanding of platelet recruitment into the lung from murine models. Here, CCL11 is linked to asthma.