In another study, the cleavage of pre-miR-143-3p is m6A-dependent, and METTL3 can positively regulate the miR-143-3p/VASH1 axis, increase lung cancer angiogenesis, and thereby regulate the VEGFA degradation and depolymerization of tubulin, playing an important role in the progress of non-small cell lung cancer (NSCLC) (128). The gene discussed is VASH1; the disease is non-small cell lung carcinoma.