We also plan to investigate the role of additional immune checkpoints such as other exhaustion markers (CTLA‐4, Tim‐3 and CD160), tumor‐associated exosomes38, 39, 40 as well as exosome‐associated immunosuppressive lipids such as phosphatidylserine41 and ganglioside GD342, 43 in the observed tumor escape in future studies. The gene discussed is CD160; the disease is neoplasm.