Ion channelopathies like α1-sodium channel subunit (SCN1A) and sodium-activated potassium channel gene (KCNT1), typically associated with Dravet syndrome and sleep-related hypermotor epilepsy, respectively, can co-occur with histologically proven FCD (Barba et al., 2014; Rubboli et al., 2019). This evidence concerns the gene KCNT1 and fleck corneal dystrophy.