A new study analyzed the causes of angiogenesis at the metabolic level, and EBV-miRNA-BART1-5p inhibited AMPKα1 and PTEN in NPC cells, thereby activating the AMPK/mTOR/HIF-1 signaling pathway, enhancing aerobic glycolysis, and promoting abnormal cell proliferation and angiogenesis [32]. The gene discussed is PRKAA1; the disease is nasopharyngeal carcinoma.