To confirm this interaction in mammalian cells, we transiently overexpressed HA‐tagged TAp63γ (HA‐TAp63γ), along with wild‐type Pin1 or its W34A mutant, in human osteosarcoma cell Saos‐2, and performed a co‐immunoprecipitation (CoIP) assay. This evidence concerns the gene PIN1 and osteosarcoma.