Using the same cell-free kit as Glover et al. (2014) but with a messenger RNA (mRNA) template as opposed to DNA, Beri et al. (2017) generated synthetic proteins, where the template was DHFR and a mutant form of superoxide dismutase (SOD1) that has been implicated in ALS, hSOD1 G93A. This evidence concerns the gene DHFR and amyotrophic lateral sclerosis.