Additional evasion mechanisms developed by tumor cells comprise the expression of suppressive molecules that inhibit NK cell function (e.g., TGFβ, prostaglandin E2 (PGE2), indoleamine 2,3-dioxygenase (IDO), adenosine and IL-10), the expression of ligands that engage NK cell inhibitory receptors and the activation of platelets that reduces NK cell activation [72,73,74,75,76]. Here, TGFB1 is linked to neoplasm.