The overexpression of miR-128 in NSCLC cells and human endothelial vein umbilical cells caused a reduction expression of VEGF-A, vascular endothelial growth factor receptor 2, and VEGFR-3, essential factors critical for cancer angiogenesis and lymphangiogenesis, and slightly declined the phosphorylation of extracellular signal-regulated kinase, phosphatidylinositol 3-kinase, and p38 signaling pathways [69]. The gene discussed is CRK; the disease is non-small cell lung carcinoma.