As shown in Table 2, the expression of COL1A2 was significantly correlated with the expression of marker genes from tumor-infiltrating B cells (CD19 and CD79A), monocytes (CD86 and CD115), TAMs (CCL2 and IL10), M1 macrophages (COX2), neutrophils (CD66b, CD11b, and CCR7), DCs (BDCA-4), T-helper (STAT4, GATA3, STAT6, BCL6, and IL17A), Treg (FOXP3, CCR8, and TGFβ), and exhausted T cells (CTLA4 and TIM-3). This evidence concerns the gene STAT4 and neoplasm.