As shown in Figures 2(g) and 2(h), the expression level of NF-κB, p-NF-κB, TLR1, TLR, TLR3, TLR4, and Myd88 was significantly promoted in the asthma group, compared with the control group, which was also inhibited by the treatment with YFP (∗∗P < 0.01 vs. control, ∗∗∗P < 0.001 vs. control, and ###P < 0.001 vs. asthma). The gene discussed is TLR3; the disease is asthma.