STAT3 codeletion in myeloid cells reversed all those changes by PDLIM2 deletion during lung tumorigenesis: elevated pulmonary recruitment of monocytes from the circulation, expanded AM differentiation from IMs and blood monocytes, heightened AM protumorigenic activation, decreased lung CTL activation, increased tumor angiogenesis and tumor cell proliferation, and reduced tumor cell apoptosis (Figure 4, C–J). The gene discussed is PDLIM2; the disease is neoplasm.