In addition to the characterized linking function of adropin 19 and fetuin 33, a recent study reported that alpha-1-microglobulin (AMBP) exacerbated inflammation and disturbed hepatic fibrotic repair after myocardial infarction through activating Akt, NF-κB, and ERK signaling and promoting macrophage migration and polarization 115. The gene discussed is AMBP; the disease is myocardial infarction.