In further support of antibody-dependent cellular phagocytosis, splenic myeloid cells from tumour-bearing (CD89-deficient) mice bound IgA through Fcα/μR (CD351) (Fig. 4c, Extended Data Fig. 6b), and killed OVCAR3 targets more effectively upon coating with TSPAN7-reactive IgA (Fig. 4d). The gene discussed is FCAMR; the disease is neoplasm.