CCL2 and endometriosis: Moreover, in lesions recovered from Ccr2−/− and Ccl2−/− mice we could still detect monocytes using immunodetection (Figs. 4 H and I and 5 G and H), indicating that there is redundancy in the CCL2–CCR2 axis in the presence of endometriosis lesions and suggesting that monocytes may also be recruited via another mechanism.