The results were partially in accordance with the mutation pattern of the subgroup of serous-like/copy number high of EC, which was characterized by mutations of TP53, PIK3CA, FBXW7, PPP2R1A, PIK3R1, CHD4, PTEN, and CSMD3 (PPP2R1A, CHD4, and CSMD3 were not included in Tumor Panel) [25]. Here, CHD4 is linked to neoplasm.