It has been hypothesized that molecular mimicry by proteins of bacteria and viruses in the human body may lead to autoimmune conditions.148 Recently, a shift in the symbiotic gut environment to a dysbiotic one has been linked to the occurrence of autoimmune conditions in humans.149 A case of molecular mimicry has been observed in the genome of Bfr, where the gene, ubb, encodes BfUbb. This evidence concerns the gene UBB and Autoimmunity.