TNNT2 and myocardial infarction: Given the higher cardiac function and lower adverse cardiac remodeling in the hearts receiving cell mixture, to investigate the possibility of whether the dual cell treatment can directly protect the myocardium from ischemic insult, we quantified the viable myocardium in the cardiac tissues harvested from all experimental groups at 8 weeks post-MI by using the cardiac troponin T (TNNT2) antibody.