Our correlation analysis data show that the CNPase+ oligodendrocyte accumulation in the peri-infarct cortex is negatively correlated with both the infarction size and severity of somatosensory-motor function (tape removal latency), suggesting that oligodendrocyte accumulation in the peri-infarct cortex is an important event during the subacute phase of stroke for limiting brain tissue loss and improving functional recovery. Here, CNP is linked to stroke disorder.