FCGR2A and aleutian mink disease: Among other parvoviruses, the highly pathogenic Aleutian mink disease parvovirus and human parvovirus B19 have, in the presence of antibodies, been shown to internalize via FcγR or the complement C1q receptor (CD93) into macrophages, endothelial cells, or B cells, resulting in the establishment of persistent infection (23, –, 26).