Moreover, MTORC1 and mTORC2 were shown to be modulators of EMT in cancer, while the silencing mTORC1 and mTORC2 resulted in an increase in E-cadherin and decrease in N-cadherin, vimentin, and snail, and other characteristics of mesenchymal to epithelial transition (MET) [38]. Here, SNAI1 is linked to cancer.