Glioblastoma cells store excess FAs into triglycerides and lipid droplets (LDs) by upregulating diacylglycerol-acyltransferase 1 (DGAT1), and inhibition of DGAT1 promotes glioblastoma cell death through excessive FAO-mediated reactive oxidative species (ROS) production [53]. Here, DGAT1 is linked to glioblastoma.