MCL1 and infarction: miR‐29a expression is also upregulated in murine heart tissues of myocardial ischemia‐reperfusion injury and overexpression of miR‐29a promotes cell apoptosis through suppression of insulin‐like growth factor I in a rat myoblast cell line169 Moreover, suppression of miR‐29a/c can reduce myocardial infarct size and IR injury‐induced cell apoptosis via the upregulation of myeloid cell leukemia 1 (MCL‐1), which is a target of miR‐29s.170