The distribution of CPT1A in tumor cells and CAFs by serial immunohistochemistry indicated that CPT1A was upregulated in CAFsPM than CAFsnPM which was identified by the a‐SMA + cell coverage (red areas), whereas expression of CPT1A was significantly lower in PM‐CRC tumor cells, compared with those in non‐PM‐CRC tumor cells(Fig. 3E and Fig. S3B). The gene discussed is ACTA1; the disease is neoplasm.