In the Netherlands, pathogenic variants in the plakophilin‐2 (PKP2) and phospholamban (PLN) gene are the most prevalent genetic predispositions for familial dilated cardiomyopathy (DCM) and arrhythmogenic cardiomyopathy (ACM) [1, 2]. This evidence concerns the gene PKP2 and familial dilated cardiomyopathy.