For example, LYL1, an oncogene that is aberrantly expressed in diverse subtypes of AML (Meng et al., 2005), assembles with MYB in leukemia cells examined in our study, similar to its functional homologue TAL1 in cases of T-ALL (Mansour et al., 2014; Sanda et al., 2012). Here, TAL1 is linked to acute myeloid leukemia.