Adjustment for WAT‐surface expression of LDLR eliminated all group‐differences in risk factors for T2D (Log10 WAT function, 1st and total DI and postprandial hypertriglyceridemia), while adjustment for WAT‐surface expression of CD36 eliminated group‐differences in all risk factors except 1st phase DI and plasma TGs at 6 h (Table 2). This evidence concerns the gene LDLR and type 2 diabetes mellitus.