Other RTKs in glioblastoma, for instance, vascular endothelial growth factor receptor (VEGFR) [18], platelet-derived growth factor receptor (PDGFR) [19] and discoidin domain RTK 1 (DDR1) [20], also led to the alterations in autophagy via the RAF/MEK [21], Akt/mammalian target of rapamycin (AKT/mTOR) [20,22] and Hypoxia-inducible factor 1/B cell lymphoma 2 (HIF-1/BCL2) signaling pathways [23,24,25]. This evidence concerns the gene KDR and glioblastoma.