To examine the issue of whether the abnormal N-glycosylation found in Rab11-deficient mice was recapitulated in DKD cells, we examined the mobility of heavily N-glycosylated proteins such as Lamp1, Lamp2, and integrin beta 1 by western blotting, which have over 10 N-glycosylation sites (33, 34). This evidence concerns the gene RAB11A and diabetic kidney disease.