Another recent study reported that administration of human placental MSC of fetal origin (hfPMSC), isolated from an unspecified region of placenta, in mice with bleomycin-induced lung fibrosis reduced collagen deposition and the production of pro-fibrotic cytokines by attenuating the dysregulation of MyD88/TGF-β signaling axis that is considered involved in the pathogenesis of pulmonary fibrosis in mice (Li et al., 2017). Here, MYD88 is linked to pulmonary fibrosis.