High expression of ACE2 in the human olfactory epithelium relative to upper airway epithelial cells may explain why COVID-19 is associated with loss of smell and suggest a potential entry point of SARS-CoV-2 into the central nervous system causing neurological symptoms in COVID-19 patients (Chen et al., 2020; Mao et al., 2020). Here, ACE2 is linked to COVID-19.