Furthermore, FLX increased the number of SOX10+ cells within the CA2-3 fields and DG and the number of SOX10+/CNPase+ cells within the CA2-3 fields of AD mice, suggesting that FLX might promote SOX10 expression on oligodendrocytes during the maturation of oligodendrocytes in the CA2-3 fields and the subsequent differentiation of mature oligodendrocytes. The gene discussed is SOX10; the disease is Alzheimer disease.