Further analysis of expression of multiple genes regulating HSC activation, cytokine production, and collagen synthesis revealed a concomitant reversal of all parameters in GdCl3 treated c-junf/f;Alb-cre mice (Fig 4B), implying a compensatory contributory role for KCs to the enhanced liver fibrosis observed in the c-junf/f;Alb-cre mice. The gene discussed is ALB; the disease is Hepatic fibrosis.