A common hallmark of endothelial dysfunction observed in many diseases is an increased expression of adhesion molecules such as ICAM‐1, VCAM‐1, P‐, and E‐selectin that may facilitate lymphocyte adhesion to activated endothelium (Deanfield et al., 2007; Kwan et al., 2007; Liao, 2013; Sena et al., 2013). The gene discussed is ICAM1; the disease is endothelial dysfunction.