Twelve weeks after the initial noise exposure, VGLUT-1 and VGLUT-2 density recovered regardless of tinnitus phenotype, but VGAT density recovery was markedly different depending on whether the animals exhibited tinnitus: exposed animals that were resistant to tinnitus showed similar or even higher levels of VGAT labeling compared to controls in every hippocampal subregion examined, while animals that developed tinnitus exhibited persistent reduction in VGAT density in the DG and areas CA3 and CA1. This evidence concerns the gene SLC17A7 and Tinnitus.