Based on the LINC01524/LOC105372666 gene mainly expressed in the human testis tissue that generate androgen, we hypothesized whether the pathogenesis of sPD was associated with the reduced secretion of androgen, as morbidity of sPD is higher in male than in female, and the partial sPD patients are accompanied with sexual dysfunction, which might be related with the abnormal expression of the LINC01524/LOC105372666 gene that contributes to the decrease in androgen level. This evidence concerns the gene LINC01524 and Platelet storage pool disease.