Finally, we downloaded the m6A-seq from shMETTL3/METTL14 and control glioma stem cells [13], and we analyzed the data concentrating on ADAR1 transcript, founding a reduction of ADAR1 methylation levels (including the ones studied in the present study, Chr1:154557253–7418) in both the shMETTL3 and shMETTL14 samples (data not shown). The gene discussed is ADAR; the disease is glioma.