Cui et al. (2017) reported that the knockout of METTL3 and METTL14 promoted the growth, self-renewal, and tumorigenesis of glioblastoma stem cells (GCS) partly by upregulating BRCA2. BRCA2 was tightly related to hereditary BC (Paul and Paul, 2014). This evidence concerns the gene METTL3 and breast cancer.