Based on the results of our differential gene expression microarray analysis of MG and non-MG (GSE72307), we investigated one of the most upregulated genes in MG, KIF26B, and demonstrated that KIF26B could promote GC proliferation and metastasis by activating the vascular endothelial growth factor (VEGF) signaling pathway (3). The gene discussed is KIF26B; the disease is gastric cancer.