PPARG and myocarditis: Overexpression of KLF5 ameliorates oxygen‒glucose deprivation- or reperfusion-induced myocardial inflammation through downregulating the levels of inflammatory cytokines (IL-1β, IL-6, IL-8, and TNF-α) and activating myocardial ischemia/reperfusion-protective proteins, peroxisome proliferator-activated receptor γ (PPARγ) and PPARγ-coactivator-1α (Li et al., 2016b).