Possible mechanisms of pathogenesis of NAFLD induced by H. pylori include (i) insulin resistance; (ii) inflammatory cytokines or adipokines, particularly CRP, TNF‐α, and IL‐6; (iii) altered lipid profile; and (iv) altered intestinal permeability and gut microbiota.42 The gene discussed is IL6; the disease is metabolic dysfunction-associated steatotic liver disease.